Mette Christine Jørgensen

Mette Christine Jørgensen

ORCID: 0000-0003-2833-7863

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  1. Transcription factors contributing to the pancreatic beta-cell phenotype

    Madsen, O. D., Jensen, J., Petersen, H. V., Pedersen, E. E., Oster, A., Andersen, F. G., Jørgensen, Mette Christine, Jensen, P. B., Larsson, L. I. & Serup, P., Jun 1997, In: Hormone and Metabolic Research. 29, 6, p. 265-70 6 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  2. Published

    The Prdm13 histone methyltransferase encoding gene is a Ptf1a-Rbpj downstream target that suppresses glutamatergic and promotes GABAergic neuronal fate in the dorsal neural tube

    Hanotel, J., Bessodes, N., Thélie, A., Hedderich, M., Parain, K., Driessche, B. V., Brandão, K. D. O., Kricha, S., Jørgensen, Mette Christine, Grapin-Botton, A., Serup, Palle, Lint, C. V., Perron, M., Pieler, T., Henningfeld, K. A. & Bellefroid, E. J., 15 Feb 2014, In: Developmental Biology. 386, 2, p. 340–357 18 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  3. Specificity of four monoclonal anti-NKx6-1 antibodies

    Klinck, R., Serup, Palle, Madsen, O. D. & Jørgensen, Mette Christine, Apr 2008, In: Journal of Histochemistry and Cytochemistry. 56, 4, p. 415-24 10 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  4. Retinoic acid signaling organizes endodermal organ specification along the entire antero-posterior axis

    Bayha, E., Jørgensen, Mette Christine, Serup, P. & Botton, A., 2009, In: PLOS ONE. 4, 6, p. e5845

    Research output: Contribution to journal › Journal article › Research › peer-review

  5. Ptf1a-mediated control of Dll1 reveals an alternative to the lateral inhibition mechanism

    Jørgensen, Mette Christine, Ahnfelt-Rønne, J., Klinck, R., Jensen, J. N., Füchtbauer, E., Deering, T., MacDonald, R. J., Wright, C. V. E., Madsen, O. D. & Serup, P., Jan 2012, In: Development. 139, 1, p. 33-45 13 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  6. Pax4 represses pancreatic glucagon gene expression

    Petersen, H. V., Jørgensen, Mette Christine, Andersen, F. G., Jensen, J., F-Nielsen, T., Jørgensen, R., Madsen, O. D. & Serup, P., Apr 2000, In: Molecular Cell Biology Research Communications. 3, 4, p. 249-54 6 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  7. Published

    Notch Controls Multiple Pancreatic Cell Fate Regulators Through Direct Hes1-mediated Repression

    de Lichtenberg, K. H., Seymour, Philip Allan, Jørgensen, Mette Christine, Kim, Y., Grapin-Botton, A., Magnuson, M. A., Nakic, N., Ferrer, J. & Serup, Palle, 2018, bioRxiv, 30 p. (bioRxiv, Developmental Biology).

    Research output: Working paper › Preprint › Research

  8. Published

    Neurog3-dependent pancreas dysgenesis causes ectopic pancreas in Hes1 mutant mice

    Jørgensen, Mette Christine, de Lichtenberg, K. H., Collin, C. A., Klinck, R., Ekberg, J. H., Engelstoft, M. S., Lickert, H. & Serup, Palle, Sep 2018, In: Development (Cambridge, England). 145, 17, p. 1-11 11 p., dev163568.

    Research output: Contribution to journal › Journal article › Research › peer-review

  9. Published

    Mind bomb 1 is required for pancreatic ß-cell formation

    Horn, S., Kobberup, S., Jørgensen, Mette Christine, Kalisz, M., Klein, T., Kageyama, R., Gegg, M., Lickert, H., Lindner, J., Magnuson, M. A., Kong, Y., Serup, Palle, Ahnfelt-Rønne, J. & Jensen, J. N., 8 May 2012, In: Proceedings of the National Academy of Sciences USA (PNAS). 109, 19, p. 7356-61 6 p.

    Research output: Contribution to journal › Journal article › Research › peer-review

  10. Published

    Jag1-Notch cis-interaction determines cell fate segregation in pancreatic development

    sgk681, sgk681, Seymour, Philip Allan, Sneppen, Kim, Trusina, Ala, Egeskov-Madsen, A. L. R., Jørgensen, Mette Christine, Jensen, Mogens Høgh & Serup, Palle, 2023, In: Nature Communications. 14, 17 p., 348.

    Research output: Contribution to journal › Journal article › Research › peer-review

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